A simple and user-friendly treatment method.

Acrochordons, also called skin tags, are small benign fibroepithelial tumors that usually attach to the skin through a thin, raised stalk, often with a visible blood vessel, known as a peduncle.¹ Skin tags may also be ”sessile”, i.e. fixed to the skin without a stalk.²

Skin tags are made up of collagen and emerge when extra cells grow in the top layers of the skin. Skin tags develop slowly over time when the skin is exposed to friction, such as between skin folds, in the axillae, on the eyelids, on the neck, below the chest, or in the groin.³

Skin tags vary in appearance from brownish to skin-coloured, and in size from less than 2 mm to 5 cm in diameter.¹ The surface may be smooth or irregular.⁴

Skin tags occur in 25-40% of the population, and the prevalence is higher in women and with increasing age.³ In addition, the prevalence is higher among obese people.² Just as diabetes is also a predisposing factor.^1,4

Some may experience itching or pain when skin tags get caught in jewelry or clothes, and trauma to the skin tag may cause bleeding due to increased vascularisation.¹ Some patients may also experience skin tags as being cosmetically unsightly.¹ The above complications are the most common reasons why people seek treatment.

Actinic keratosis, also called solar keratosis, is considered to be premalignant skin changes, primarily in the basal cell layer of the epidermis¹.

Among the causes for this development is long-term exposure to sun throughout life, and about 80% of all actinic keratoses are found in sun-damaged skin areas².

Actinic keratoses are often located on the head, scalp, ears, and neck, on the back of your hand and the back of upper and lower extremities^3,1. Actinic keratoses manifest as small, adherent pink and yellowish-brown scales. They vary in thickness from atrophic to hypertrophic, and in size from 1mm to several centimetres¹.

In clinical terms, actinic keratoses are divided into three severity grades^1,4.

Early-stage actinic keratoses may itch slightly. When the lesions become hyperkeratotic, they may cause both functional and cosmetic impairment², and patients often seek treatment.

Until recently, it was assumed that grade III actinic keratoses were more dysplastic than grade II actinic keratoses. However, recent research indicates that both grades of actinic keratoses have the same level of dysplasia, which emphasizes that all actinic keratoses should be treated, irrespective of their thickness⁵.

If left untreated, actinic keratoses can penetrate the dermis, allowing the premalignant state to develop into non-melanoma skin cancer in the form of basal cell carcinoma (BCC) and particularly squamous cell carcinoma (SCC) of the skin⁴. However, timely treatment with cryosurgery can prevent this development.

Cervical contact bleeding is bleeding that originates from the cervix. There are several potential causes for bleeding from the cervix, and women are usually encouraged to go to a gynecologist for evaluation if they experience cervical bleeding. Cervical bleeding after copulation or another contact with the cervix is relatively common and may result from a wide range of non-cancer-related pathologies, such as cervical polyps, various metaplastic states^{1, 2} ectopic/erosions^3, and cervicitis.⁴

Common warts, also known as verruca vulgaris, are superficial viral infections of the skin caused by human papilloma virus (HPV).

Common warts may vary in appearance, but frequently occur as multiple, hyperkeratotic cauliflower-like papules on the skin. They most often range from 1-10 mm in diameter.¹ The surface of the common wart has small black dots, sometimes invisible to the naked eye, representing blood clots in the capillaries of the wart. These capillaries can contribute to the diagnosis of a common wart, as this clinical observation is not seen in differential diagnoses such as callosities or soft tissue sarcomas. In addition, the patient’s fingerprint will not be visible on the surface of a common wart as is typically the case for callous skin, for instance.²

Common warts occur in places where the skin has been exposed to abrasions or other microtrauma, as the virus is inoculated into the skin through the epithelial tissue and penetrates further down into the deeper epithelial layers.³ HPV is resistant and spreads through both direct and indirect contact, leaving a large proportion of everyday objects as reservoirs of infection. Particularly in case of periungual warts, located around nails and nail walls, there is an increased risk of spread to the patient’s lips, oral cavity and tongue if the patient is a nailbiter.⁴ In addition, visible common warts may be perceived as socially unacceptable, as described in several studies. The fear of passing on infection may result in social isolation for the common wart patient, thus potentially affecting the patient’s mental health.^1,5 Due to their long incubation time of 1-6 months, it is beneficial to treat warts in order to better break the chain of infection and reduce the infection of others.

Genital warts or Condyloma Acuminatum are the anogenital expression of HPV and is one of the most common sexually transmitted diseases.^{1, 2}

Gingival Melanin Pigmentation (GMP), which is benign however a cosmetic concern, is a focal pigmentation of endogenous origin that occurs because of the excessive deposition of melanin. The color of the oral melanin pigmentation may vary from light to dark brown or black, depending on the amount and localization in the tissue.¹

Seborrheic keratosis (also known as Seborrheic verruca and Senile wart) is a wart-like noncancerous benign skin growth that originates in keratinocytes.^{1, 2, 3}

Lentigo solaris also known as “lentigo senillis”, “liver spots” and “age spots”, are benign changes on the skin that include hyperplasia of the epidermis and increased pigmentation of the basal layer1.

The changes on the skin occur due to sun exposure, and thus appear on sun-exposed areas of the body, such as the face, backs of the hands, shoulders, and back^{1, 2}. The changes can develop slowly over years, or occur suddenly.^{1, 2}

Lentigo solaris varies in appearance, from light brown to dark or black spots on the skin, from oval to irregular, and in some cases nodular in the lesion or slightly scaly. They also vary in size, from a few millimeters to several centimeters in diameter. The changes often become darker and larger proportionally with age^{2, 3}. Lentigo occurs on sun-exposed skin, and most frequently among light-skinned people,

The incidence of lentigo solaris is higher in fair-skinned people who have been exposed to a lot of sun².

Children with the rare, inherited skin disease xeroderma pigmentosum develop lentigo solaris in the first months of life, even after minimal sun exposure⁴.

Lentigo solaris can be cosmetically disfiguring for some patients, which is why they seek treatment.

Molluscum contagiosum, also called water warts, is a viral infection caused by a DNA poxvirus manifesting in the skin.^1,2 Molluscum contagiosum is one of the five most prevalent skin diseases worldwide.²

Transmitted by direct skin-to-skin contact,² molluscum contagiosum develops slowly over weeks. As they grow, the mollusca develop a central dimple, also known as umbilication.¹

Molluscum contagiosum presents as dome-shaped papules containing cheesy material, the molluscum body. The mollusca have a smooth surface and vary in appearance from pale red to skin-coloured and in size from 3-10 mm.^1,2

Molluscum contagiosum is most prevalent in children (about 9 out of 10 cases) of all ages, sexually active persons and in immunosuppressed individuals ^1,2. The prevalence is also higher among atopics^1,2 and in countries with a warm climate.²

In children, the mollusca are typically located on the extremities, trunk and face, often in body creases. Sexually transmitted molluscum contagiosum is located in the lower abdomen and the genital region. In immunosuppressed individuals, molluscum contagiosum more frequently presents as large, multiple mollusca.¹

The incubation period is estimated to be 2-8 weeks,² which means that molluscum contagiosum should be treated to break the chain of infection and reduce the number of further infectees. In some persons, molluscum contagiosum may also cause itching, eczema and secondary bacterial infections.^1,2 Some patients may also experience molluscum contagiosum as being cosmetically unsightly. The above complications are the most common reasons why people seek treatment.

Warts and verrucas are caused by a viral infection of the skin with a special variant of human papillomavirus (HPV).

Patients may perceive warts and verrucas as socially unacceptable if they occur in particularly visible places such as face or hands. This may result in social isolation affecting the patient’s mental health.

Verrucas on the sole of the foot are pressed flat during the daily mobilisation and grow inwards. As the verruca pushes into the sole of the foot, its hyperkeratotic, rough surface may be a source of pain that restricts mobilisation.

A shared feature of warts and verrucas is that the virus is highly resistant and spreads through both direct and indirect contact, leaving a large proportion of the population and everyday objects as reservoirs of HPV infection. Although HPV persists in the skin after treatment of the actual wart or verruca, treatment can prevent the spread of the infection.

As the incubation period is 1-6 months, warts and verrucas on your hands and feet should therefore be treated immediately to break the infection chain and reduce infection of others.^{1, 2}

Warts and verrucas are caused by a viral infection of the skin with a special variant of human papillomavirus (HPV).

Patients may perceive warts and verrucas as socially unacceptable if they occur in particularly visible places such as face or hands. This may result in social isolation affecting the patient’s mental health.

Verrucas on the sole of the foot are pressed flat during the daily mobilisation and grow inwards. As the verruca pushes into the sole of the foot, its hyperkeratotic, rough surface may be a source of pain that restricts mobilisation.

A shared feature of warts and verrucas is that the virus is highly resistant and spreads through both direct and indirect contact, leaving a large proportion of the population and everyday objects as reservoirs of HPV infection. Although HPV persists in the skin after treatment of the actual wart or verruca, treatment can prevent the spread of the infection.

As the incubation period is 1-6 months, warts and verrucas on your hands and feet should therefore be treated immediately to break the infection chain and reduce infection of others.^{1, 2}

Warts and verrucas are caused by a viral infection of the skin with a special variant of human papillomavirus (HPV).

Patients may perceive warts and verrucas as socially unacceptable if they occur in particularly visible places such as face or hands. This may result in social isolation affecting the patient’s mental health.

Verrucas on the sole of the foot are pressed flat during the daily mobilisation and grow inwards. As the verruca pushes into the sole of the foot, its hyperkeratotic, rough surface may be a source of pain that restricts mobilisation.

A shared feature of warts and verrucas is that the virus is highly resistant and spreads through both direct and indirect contact, leaving a large proportion of the population and everyday objects as reservoirs of HPV infection. Although HPV persists in the skin after treatment of the actual wart or verruca, treatment can prevent the spread of the infection.

As the incubation period is 1-6 months, warts and verrucas on your hands and feet should therefore be treated immediately to break the infection chain and reduce infection of others.^{1, 2}